Lung cancer happening mechanism pursuit | treatments for lung ...

??The university of Texas southwestern medical center JohnMinna of, and colleagues recently published in the journal cancer research articles, they pointed out that in the study found that the cancer early lung cells form a chromosome 3 p, part of the regional (3 p21. 3) often lack.Researchers think that this is a easy to cause cancer response of mutations fragile site, is a visible at very early cancer and the development of carcinoma in situ in the change.This phenomenon in without cancer of smokers in the lung cells can also be seen.
??
??The results also suggest that chromosome 3 p area is lung cancer cells in the process of the development of the earliest is detected a genetic changes area.This discovery for patients with lung cancer, said the gene for early detection and treatment.
??
??3 p area contains many tumor-suppressor genes (including FUS1) not only can inhibit tumor growth and transfer, and can induce human lung cancer cells to die.Although the area is located in the 3 p FUS1 tumor-suppressor genes in the cellular canceration process can play effective function, but it is puzzled, so far in the cancer cell?..s have not yet found the genes of any a kind of protein products.
??
??Southwestern medical center scientists the study found that, although FUS1 gene guidance, but protein synthesis protective tumor cells appear in some of the abnormal situation will lead to the gene guidance in the synthesis of suppressor proteins lose activity.
??
??The researchers introduces, in normal circumstances, the fatty acids in cells chain and FUS1 gene guidance synthetic suppressor proteins connection in the cell membrane surface adhesion.But in some cancer cells can appear is called a (14 acylation process of missing, fatty acid in cells is not with FUS1 chain guide synthetic suppressor proteins connection, make suppressor proteins lose activity, and in a certain degree to continue to cancer development.
??
??In the study, professor of LinJi pointed out that the results suggest that, the 14 ( cells acylation process for the lack of cancer may be contributing to the occurrence and development of potential root causes.
??
??The world famous lung cancer gene therapy JackRoth experts to think, FUS1 dr guidance of tumor suppressor proteins produced the function lack of lung cancer may be an early change, and also may occur in other cancer (such as breast and kidney cancer).The research put forward two potential treatment strategies: one is to restore the tumor cells 14 silane acylation\The other is a FUS1 activity will gene into tumor cells to gene therapy.
??
??Roth explained that, for tumor cells of the replacement of a FUS1 gene treatment strategies may help prevent or delay the occurrence of lung cancer.He suggested that, for the three p21. 3 area missing patients, shall conduct therapeutic FUS1 genes as early as possible replacement.
??
??According to information, the American Anderson cancer center researchers are currently on this one
??The university of Texas southwestern medical center JohnMinna of, and colleagues recently published in the journal cancer research articles, they pointed out that in the study found that the cancer early lung cells form a chromosome 3 p, part of the regional (3 p21. 3) often lack.Researchers think that this is a easy to cause cancer response of mutations fragile site, is a visible at very early cancer and the development of carcinoma in situ in the change.This phenomenon in without cancer of smokers in the lung cells can also be seen.
??
??The results also suggest that chromosome 3 p area is lung cancer cells in the process of the development of the earliest is detected a genetic changes area.This discovery for patients with lung cancer, said the gene for early detection and treatment.
??
??3 p area contains many tumor-suppressor genes (including FUS1) not only can inhibit tumor growth and transfer, and can induce human lung cancer cells to die.Although the area is located in the 3 p FUS1 tumor-suppressor genes in the cellular canceration process can play effective function, but it is puzzled, so far in the cancer cells have not yet found the genes of any a kind of protein products.
??
??Southwestern medical center scientists the study found that, although FUS1 gene guidance, but protein synthesis protective tumor cells appear in some of the abnormal situation will lead to the gene guidance in the synthesis of suppressor proteins lose activity.
??
??The researchers introduces, in normal circumstances, the fatty acids in cells chain and FUS1 gene guidance synthetic suppressor proteins connection in the cell membrane surface adhesion.But in some cancer cells can appear is called a (14 acylation process of missing, fatty acid in cells is not with FUS1 chain guide synthetic suppressor proteins connection, make suppressor proteins lose activity, and in a certain degree to continue to cancer development.
??
??In the study, professor of LinJi pointed out that the results suggest that, the 14 ( cells acylation process for the lack of cancer may be contributing to the occurrence and development of potential root causes.
??
??The world famous lung cancer gene therapy JackRoth experts to think, FUS1 dr guidance of tumor suppressor proteins produced the function lack of lung cancer may be an early change, and also may occur in other cancer (such as breast and kidney cancer).The research put forward two potential treatment strategies: one is to restore the tumor cells 14 silane acylation\\The other is a FUS1 activity will gene into tumor cells to gene therapy.
??
??Roth explained that, for tumor cells of the replacement of a FUS1 gene treatment strategies may help prevent or delay the occurrence of lung cancer.He suggested that, for the three p21. 3 area missing patients, shall conduct therapeutic FUS1 genes as early as possible replacement.
??
??According to information, the American Anderson cancer center researchers are currently on this one
??Therapy in phase I clinical trials.In order to formulate correct treatment, the team after DuoNian efforts have joining together with lung cancer cells change the map.
??
??Roth, dr Anderson cancer center introduced the researchers found a gene therapy is easy to be used, tumor cells absorb fat bubble package millions of gene copies FUS1 gene therapy.At present there have been six cases of metastatic lung cancer patients received the gene therapy.

Source: http://treatments-for-lung-cancer.com/Lung-cancer-happening-mechanism-pursuit-2048/

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